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Jay Michael Balagtas, MD

  • Jay Michael S Balagtas
  • “I value the bonds I form not only with the patient but with the family as a whole.”

My philosophy has always been to seek out the best possible medical therapies for a patient's disease. Even in cases where we can't provide a definitive cure, we can still assist the family and patient in other ways. I let parents know that my care team and I will walk the whole way with them.

My patients are very inspiring to me. No matter how sick they are, they always put a smile on my face. Even though I am a pediatrician, I value the bonds I form not only with the patient but with the family as a whole. My care team and I always consider the needs of the entire family when we are designing treatment approaches and actively seek ways to reduce the burden on a family caring for a chronically ill child.

Especialidades

Hematology-Oncology

Trabajo y Educación

Formación Profesional

New York Medical College Registrar, Valhalla, NY, 5/31/1998

Internado

United States Naval Medical CenterSan Diego, San Diego, CA, 6/30/1999

Residencia

United States Naval Medical CenterSan Diego, San Diego, CA, 06/30/2003

Compañerismo

Lucile Packard Children's Hospital, Palo Alto, CA, 6/30/2009

Certificaciones Médicas

Pediatric Hematology-Oncology, American Board of Pediatrics

Pediatrics, American Board of Pediatrics

Todo Publicaciones

Subcutaneous Panniculitis-Like T-Cell Lymphoma: Pediatric Case Series Demonstrating Heterogeneous Presentation and Option for Watchful Waiting PEDIATRIC BLOOD & CANCER Johnston, E. E., LeBlanc, R. E., Kim, J., Chung, J., Balagtas, J., Kim, Y. H., Link, M. P. 2015; 62 (11): 2025-2028

View details for DOI 10.1002/pbc.25626

View details for Web of Science ID 000364581100029

View details for PubMedID 26146844

Breakpoint analysis of transcriptional and genomic profiles uncovers novel gene fusions spanning multiple human cancer types. PLoS genetics Giacomini, C. P., Sun, S., Varma, S., Shain, A. H., Giacomini, M. M., Balagtas, J., Sweeney, R. T., Lai, E., Del Vecchio, C. A., Forster, A. D., Clarke, N., Montgomery, K. D., Zhu, S., Wong, A. J., van de Rijn, M., West, R. B., Pollack, J. R. 2013; 9 (4)

View details for DOI 10.1371/journal.pgen.1003464

View details for PubMedID 23637631

Therapeutic Complications in a Patient With High-Risk Acute Lymphoblastic Leukemia and Undiagnosed Hereditary Hemochromatosis PEDIATRIC BLOOD & CANCER Balagtas, J. M., Dahl, G. V. 2012; 58 (1): 101-103

Abstract

Hereditary hemochromatosis (HH) is an autosomal-recessive disorder of iron metabolism that most commonly manifests in the fourth or fifth decade of life. Here, we describe a 14-year-old male who presented with high-risk acute lymphoblastic leukemia and previously undiagnosed HH. His treatment course was remarkable for significant therapeutic complications, including iron overload, hepatic failure, cardiac dysfunction, and death. Postmortem testing revealed homozygosity for the C282Y mutation, confirming the diagnosis of HH. Since HH mutations occur commonly in select populations, screening patients with leukemia for HH may better inform treatment decisions regarding chemotherapy, transfusions, and/or iron chelation therapy.

View details for DOI 10.1002/pbc.22829

View details for Web of Science ID 000297641300020

View details for PubMedID 22076832

Liposomal amphotericin B associated with severe hyperphosphatemia PEDIATRIC INFECTIOUS DISEASE JOURNAL Sutherland, S. M., Hong, D. K., Balagtas, J., Gutierrez, K., Dvorak, C. C., Sarwal, M. 2008; 27 (1): 77-79

Abstract

We report 4 patients who developed hyperphosphatemia while receiving liposomal amphotericin B to treat an invasive fungal infection. Resolution of the hyperphosphatemia occurred after transition to amphotericin B lipid complex. This phenomenon may occur more commonly in patients with mild to moderate renal insufficiency.

View details for DOI 10.1097/INF.0b013e31815922a3

View details for Web of Science ID 000252076200019

View details for PubMedID 18162947